Placental surface area mediates the association betweenmethylation in placenta and full-term low birth weight in girls.

View Abstract

Background

Fibroblast growth factor receptor 2 () gene encodes a protein of the fibroblast growth factor receptor family.gene expression is associated with the regulation of implantation process of placenta which plays a vital role in fetal growth. DNA methylation is widely known as a mechanism of fetal growth. However, it is unclear whether and how DNA methylation ofgene in the placenta is associated with full-term low birth weight. This case-control study aims to explore the links betweenmethylation in placenta and full-term low birth weight and to further examine the mediation effect of placental surface area on this association.

Results

We conducted analyses for each of the five valid CpG sites atin 165 mother-baby pairs (86 FT-LBW vs. 79 FT-NBW) and found that per one standard deviation increase in the DNA methylation of CpG 11 atwas associated with 1.64-fold higher risk of full-term low birth weight (OR = 1.64, 95% CI = [1.07, 2.52]) and 0.18 standard deviation decrease in placental surface area ( = - 0.18; standard error = 0.08, = 0.02). The mediation effect of placental surface area on the association between DNA methylation and full-term low birth weight was significant in girls (OR = 1.38, 95% CI = [1.05, 1.80]) but not in boys. The estimated mediation proportion was 48.38%.

Conclusion

Our findings suggested that placental surface area mediated the association between DNA methylation ofin placenta and full-term low birth weight in a sex-specific manner. Our study supported the importance of placental epigenetic changes in placental development and fetal growth.

Investigators
Abbreviation
Clin Epigenetics
Publication Date
2018-03-22
Volume
10
Page Numbers
39
Pubmed ID
29588807
Medium
Electronic-eCollection
Full Title
Placental surface area mediates the association betweenmethylation in placenta and full-term low birth weight in girls.
Authors
Tian FY, Wang XM, Xie C, Zhao B, Niu Z, Fan L, Hivert MF, Chen WQ