Glycation of fetal hemoglobin reflects hyperglycemia exposure in utero.

View Abstract

OBJECTIVE

The lifetime risk of metabolic diseases in offspring of women with gestational diabetes mellitus (GDM) depends, at least in part, on the impact of glycemic fetal programming. To quantify this impact, we have developed and validated a unique mass spectrometry method to measure the percentage of glycated hemoglobin in cord blood.

RESEARCH DESIGN AND METHODS

This case-control study includes 37 GDM women and 30 pregnant women with normal glucose tolerance (NGT).

RESULTS

Glycation of the α-chain (Glα) was higher in neonates from GDM (2.32 vs. 2.20%, P < 0.01). Glα strongly correlated with maternal A1C measured at delivery in the overall cohort (r = 0.67, P < 0.0001) as well as in each group (GDM: r = 0.66, P < 0.0001; NGT: r = 0.50, P = 0.01).

CONCLUSIONS

Thus, Glα may reflect hyperglycemic exposure during the last weeks of fetal development. Future studies will confirm Glα is a predictive biomarker of prenatally programmed lifetime metabolic health and disease.

Investigators
Abbreviation
Diabetes Care
Publication Date
2014-07-10
Volume
37
Issue
10
Page Numbers
2830-3
Pubmed ID
25011951
Medium
Print-Electronic
Full Title
Glycation of fetal hemoglobin reflects hyperglycemia exposure in utero.
Authors
Dupont FO, Hivert MF, Allard C, Ménard J, Perron P, Bouchard L, Robitaille J, Pasquier JC, Auray-Blais C, Ardilouze JL